🧬

Age & Use Verification

You must be 21+ to enter this site.

All products discussed on this site are Research Use Only (RUO) compounds intended strictly for laboratory research. Nothing here is medical advice, and these products are not for human or veterinary use.

Exit

We use cookies for analytics and to improve your experience. See our disclosure.

Ac-SDKP: Research Profile, Dosages & Supplier Prices

Anti-fibrotic research · Lyophilized powder · Also known as: N-acetyl-Ser-Asp-Lys-Pro, Goralatide-class tetrapeptide

Ac-SDKP is a anti-fibrotic research compound with a molecular weight of 487.5 g/mol and a research half-life of ~4-5 minutes (cleaved by ace). Ac-SDKP is a naturally occurring tetrapeptide released from thymosin beta-4 by prolyl oligopeptidase, and destroyed within minutes by angiotensin-converting enzyme (ACE). Despite its fleeting half-life it is a significant endogenous regulator: a hematopoietic stem cell inhibitor and, more prominently in current research, an anti-fibrotic and anti-inflammatory signal in heart, kidney and lung tissue.

Research Use Only (RUO). Not for human or veterinary use. Not medical advice.

SequenceAc-Ser-Asp-Lys-Pro-OH
FormulaC20H33N5O9
Mol. weight487.5 g/mol
Half-life~4-5 minutes (cleaved by ACE)
Forms soldLyophilized powder

What is Ac-SDKP?

Research interest surged with the recognition that part of the organ-protective benefit of ACE inhibitors may come from raising Ac-SDKP levels - blocking its breakdown. Animal studies show Ac-SDKP reduces cardiac and renal fibrosis, macrophage infiltration and TGF-beta signaling. It is also the C-terminal cleavage product released from TB-500's parent molecule, linking it to the broader thymosin beta-4 research story.

Mechanism of Action (Research Context)

Anti-fibrotic signaling: suppresses TGF-beta/Smad-driven collagen deposition, inhibits macrophage and myofibroblast activation, and inhibits hematopoietic stem cell proliferation via cell-cycle arrest. Rapidly inactivated by ACE N-domain cleavage.

Research Areas

Cardiac and renal fibrosisTGF-beta signalingMacrophage-driven inflammationHematopoietic stem cell regulationACE inhibitor mechanism research

Ac-SDKP Dosages in Research

5mg10mg vials

Animal fibrosis studies typically use continuous infusion (osmotic minipumps) because of the ~4-minute half-life. Research-context only.

Dosage information is a summary of published laboratory research and is provided for informational purposes only. These compounds are not for human use.

Storage & Handling

Lyophilized: -20C desiccated. Reconstituted: 2-8C, use promptly; short plasma half-life reflects enzymatic rather than solution instability.

Ac-SDKP Supplier Price Comparison

No tracked suppliers stock Ac-SDKP yet — inventory syncs run continuously. Check our supplier reviews.

Ac-SDKP FAQ

A natural tetrapeptide (N-acetyl-Ser-Asp-Lys-Pro) released from thymosin beta-4, studied mainly for anti-fibrotic and anti-inflammatory effects in heart, kidney and lung.
ACE degrades Ac-SDKP within minutes. ACE inhibitors raise Ac-SDKP levels substantially, and animal studies suggest this contributes to their organ-protective, anti-fibrotic effects.
98%+ HPLC purity and mass spec identity at 487.5 Da. It is a small, cheap tetrapeptide - substitution is rare but identity confirmation is trivial to demand.
🧬

Before you go —

Get weekly supplier alerts, price-drop notifications, and COA updates. Plus our featured pick: Certified Pep.